
Ira A. Tabas MD
Diabetes, Lipid Metabolism
Professor, Medicine, Columbia University College of Phys & Surg
622 W 168th StNew York, NY 10032
Overview of Dr. Tabas
Dr. Ira Tabas is an endocrinologist in New York, NY and is affiliated with multiple hospitals in the area, including New York-Presbyterian Hospital and NewYork-Presbyterian/Columbia University Irving Medical Center. He received his medical degree from Washington University in St. Louis School of Medicine and has been in practice 36 years. He is one of 144 doctors at New York-Presbyterian Hospital and one of 56 doctors at NewYork-Presbyterian/Columbia University Irving Medical Center who specialize in Endocrinology. He has more than 100 publications and over 500 citings.
Education & Training
- New York Presbyterian Hospital (Columbia Campus)Residency, Internal Medicine, 1981 - 1983
- Washington University in St. Louis School of MedicineClass of 1981
Certifications & Licensure
- NY State Medical License 1982 - 2027
- American Board of Internal Medicine Internal Medicine
- American Board of Internal Medicine Endocrinology, Diabetes and Metabolism
Awards, Honors, & Recognition
- Elected Member The American Society for Clinical Investigation, 1992
Publications & Presentations
PubMed
- Location, location, location: Cholesterol in lipid droplets as a driver of MASH progression.Xiaobo Wang, Ira Tabas
Proceedings of the National Academy of Sciences of the United States of America. 2025-06-10 - 1 citationsLow MBOAT7 expression, a genetic risk for MASH, promotes a profibrotic pathway involving hepatocyte TAZ upregulation.Mary P Moore, Xiaobo Wang, John Paul Kennelly, Hongxue Shi, Yuki Ishino
Hepatology. 2025-02-01 - 4 citationsInflammatory crosstalk impairs phagocytic receptors and aggravates atherosclerosis in clonal hematopoiesis in mice.Wenli Liu, Brian D Hardaway, Eunyoung Kim, Jessica Pauli, Justus Leonard Wettich
The Journal of Clinical Investigation. 2024-11-12
Journal Articles
- JACC Focus Seminar: Macrophage in CVD Series JACC Focus Seminar Macrophage Trafficking, Inflammatory Resolution, and Genomics in Atherosclerosis: JACC Macrophage in CV...Amanda C Doran, Jason C Kovacic, Edward A Fisher, Ira Tabas, ScienceDirect
- A New Activator of Hepatocyte CaMKII in Fasting and Type 2 DiabetesIra Tabas, MD, Diabetes Care
Press Mentions
- Having This Body Shape Means You’re More at Risk for Colorectal Cancer, Diabetes and Heart DiseaseJanuary 17th, 2025
- 'Stench of Death' Compound Helps Your Body Feast on Its Own Rotting CellsJanuary 31st, 2020
- Putrid Compound Putrescine May Have a Sweet Side Gig as Atherosclerosis TreatmentJanuary 31st, 2020
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Grant Support
- Discovery of novel therapeutic targets for NASH through deep phenotyping of human knockouts and mechanistic studiesCOLUMBIA UNIVERSITY HEALTH SCIENCES2022–2026
- Discovery of novel therapeutic targets for NASH through deep phenotyping of human knockouts and mechanistic studiesCOLUMBIA UNIVERSITY HEALTH SCIENCES2022–2026
- Discovery of novel therapeutic targets for NASH through deep phenotyping of human knockouts and mechanistic studiesCOLUMBIA UNIVERSITY HEALTH SCIENCES2022–2026
- Discovery of novel therapeutic targets for NASH through deep phenotyping of human knockouts and mechanistic studiesCOLUMBIA UNIVERSITY HEALTH SCIENCES2022–2026
- Discovery of novel therapeutic targets for NASH through deep phenotyping of human knockouts and mechanistic studiesCOLUMBIA UNIVERSITY HEALTH SCIENCES2022–2026
- A Mechanistic and Translational Research Program Linking Impaired Resolution, Defective Efferocytosis, and Clonal Hematopoiesis to the Formation of Clinically Dangerous Atherosclerotic PlaquesCOLUMBIA UNIVERSITY HEALTH SCIENCES2019–2026
- Training in Cardiovascular Translational ResearchCOLUMBIA UNIVERSITY HEALTH SCIENCES2014–2024
- Mechanisms Of Defective Efferocytosis In AtherosclerosisNational Heart, Lung, And Blood Institute2012
- Autophagy In Advanced AtherosclerosisNational Heart, Lung, And Blood Institute2011–2012
- The Unfolded Protein Response In Sterol CytoxicityNational Heart, Lung, And Blood Institute2005–2012
- Role Of Camkii In Hepatic GluconeogenesisNational Center For Research Resources2011
- Molecular Links Between Insulin Resistance And Macrophage Death In AtherosclerosiNational Heart, Lung, And Blood Institute2007–2011
- Mechanisms Of Atherogenesis In Insulin ResistanceNational Heart, Lung, And Blood Institute2007–2011
- Administrative CoreNational Heart, Lung, And Blood Institute2007–2011
- 2010 Lipoprotein Metabolism GrcNational Heart, Lung, And Blood Institute2010
- Macrophage Death In Plaque VulnerabilityNational Heart, Lung, And Blood Institute2006–2010
- Interaction Of Atherogenic Lipoproteins With MacrophagesNational Heart, Lung, And Blood Institute1997–2010
- The Unfolded Protein Response In Sterol CytotoxicityNational Heart, Lung, And Blood Institute2004–2007
- Core D--PathologyNational Heart, Lung, And Blood Institute2004–2006
- Spingomyelinase In Atherosclerosis And Foam Cell BiologyNational Heart, Lung, And Blood Institute2002–2006
- Macrophage Death In Plaque VulnerabilityNational Heart, Lung, And Blood Institute2005
- Conference On Cellular Biology Of AtherosclerosisNational Heart, Lung, And Blood Institute2005
- Macrophage Death And Lipid Metabolism In AtherosclerosisNational Heart, Lung, And Blood Institute2000–2002
- Form Cell APO (A)/Lp(A) Receptor--Cloning And FunctionNational Heart, Lung, And Blood Institute2000–2002
- Core--PathologyNational Heart, Lung, And Blood Institute1997–2002
- Arterial Sphingomyelinase, Proteoglycans And AtherogenesisNational Heart, Lung, And Blood Institute1997–2001
- Foam Cell APO (A)/Lp(A) Receptor--Cloning And FunctionNational Heart, Lung, And Blood Institute1999
- Macrophage Cholesterol And Phospholipid Metabolism--Foam Cell NecrosisNational Heart, Lung, And Blood Institute1997–1999
- Macrophage Cholesterol And Phospholipid Metabolism --Foam Cell NecrosisNational Heart, Lung, And Blood Institute1996
- ACAT Reactivity In MacrophagesNational Heart, Lung, And Blood Institute1995–1996
- ACAT Reactivity In MacrophagesNational Heart, Lung, And Blood Institute1988–1994
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