
Jane Sibley Titterington MD PhD
Heart Failure & Transplantation
Cardiology Fellow
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4000 Cambridge StSte G600Kansas City, KS 66160
Phone+1 913-588-9700
Fax+1 913-588-9770
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Education & Training
Emory University School of MedicineFellowship, Advanced Heart Failure and Transplant Cardiology, 2017 - 2018
Emory University School of MedicineFellowship, Cardiovascular Disease, 2012 - 2017
Emory University School of MedicineResidency, Internal Medicine, 2010 - 2013
Tulane University School of MedicineClass of 2010
Kansas State UniversityB.A., Microbiology major, Spanish minor, 1998 - 2002
Certifications & Licensure
KS State Medical License 2018 - 2023
MO State Medical License 2018 - 2023
GA State Medical License Active through 2018
Awards, Honors, & Recognition
- Haymaker Award Kansas State University Division of Biology, 2002
- Phi Beta Kappa 2002
- Research Award Kansas State University Center for Basic Cancer, 2001
Publications & Presentations
PubMed
- 28 citationsNovel PET and Near Infrared Imaging Probes for the Specific Detection of Bacterial Infections Associated With Cardiac Devices.Kiyoko Takemiya, Xinghai Ning, Wonewoo Seo, Xiaojian Wang, Rafi Mohammad
JACC. Cardiovascular Imaging. 2019-05-01 - 4 citationsGender differences in acute coronary syndromes: focus on the women with ACS without an obstructing culprit lesion.Jane S Titterington, Olivia Y. Hung, Anita Saraf, Nanette K. Wenger
Expert Review of Cardiovascular Therapy. 2018-03-07 - 2 citationsEvolving cardiovascular care for women: a decade of progressOlivia Y. Hung, Jane S Titterington, Nanette K. Wenger
Future Cardiology. 2015-05-29
Abstracts/Posters
- Growth hormone-releasing peptide-2 reduces oxidative stress in ApoE -/- mice but does not reduce atherosclerosis.Titterington JS, Sukhanov S, Vaughn CE, Delafontaine P, FASEB J, 1/1/2009
- Insulin-like growth factor I downregulates lipoprotein lipase and suppresses atherosclerotic foam cell formation in vivo and in vitro.Sukhanov S. Vaughn CE, Higashi Y, Titterington JS, Delafontaine P, FASEB J, 1/1/2009
- Chronic Left-Ventricular Dysfunction Increases Oxidative Stress in Skeletal Muscle: Potential Mechanism of Cardiac Cachexia.Semprun-Prieto L, Sukhanov S, Bhutto J, Titterington JS, Delafontaine P, FASEB J, 1/1/2009
Lectures
- Growth hormone-releasing peptide-2 reduces oxidative stress in ApoE -/- mice but does not reduce atherosclerosis.Experimental Biology - 4/1/2009
- Insulin-like growth factor I downregulates lipoprotein lipase and suppresses atherosclerotic foam cell formation in vivo and in vitro.Experimental Biology - 4/1/2009
- Chronic Left-Ventricular Dysfunction Increases Oxidative Stress in Skeletal Muscle: Potential Mechanism of Cardiac Cachexia.Experimental Biology - 4/1/2009
Press Mentions
Resveratrol-Mediated Expression of KLF15 in the Ischemic Myocardium Is Associated with an Improved Cardiac PhenotypeJanuary 7th, 2017
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