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Vinicio De Jesus Perez, MD, Pulmonology, Palo Alto, CA

Vinicio Alberto De Jesus Perez MD

Critical Care Medicine, Pleural Disease


Assistant Professor, Medicine, Stanford University School of Medicine

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  • 300 Pasteur DrPalo Alto, CA 94304

  • Phone+1 650-723-4000

  • Fax+1 650-723-6272

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Education & Training

  • Stanford Health Care-Sponsored Stanford University
    Stanford Health Care-Sponsored Stanford UniversityFellowship, Pulmonary Disease and Critical Care Medicine, 2004 - 2006
  • Mass General Brigham/Massachusetts General Hospital
    Mass General Brigham/Massachusetts General HospitalResidency, Internal Medicine, 2000 - 2003
  • University of Puerto Rico School of Medicine
    University of Puerto Rico School of MedicineClass of 2000
  • University of Puerto Rico, Rio Piedras Campus
    University of Puerto Rico, Rio Piedras CampusB.S., Biology, 1992 - 1996

Certifications & Licensure

  • CA State Medical License
    CA State Medical License 2004 - 2026
  • Critical Care Medicine
    American Board of Internal Medicine Critical Care Medicine
  • Pulmonary Disease
    American Board of Internal Medicine Pulmonary Disease

Awards, Honors, & Recognition

  • Early Career award ATS Pulmonary Circulation Assembly, 2016
  • Young Physician Scientist Award American Society of Clinical Investigation, 2016
  • Dorothy Dee and Marjorie Helene Boring Trust Award 2016
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Publications & Presentations

PubMed

Journal Articles

  • Molecular pathogenesis and current pathology of pulmonary hypertension  
    VA de Jesus Perez, Heart Fail Rev, 1/22/2015
  • Assessment of pulmonary arterial wall anatomy in humans and pig (sus scrofa) using optical coherence tomography  
    NW Brunner, RT Zamanian, F Ikeno, Y Mitsutake, AJ Connolly, E Shuffle, K Yuan, M Orcholski, J Lyons and VA de Jesus Perez, Comparative Medicine, 1/1/2015
  • Loss-of BMPR2 is associated with abnormal DNA repair in pulmonary arterial hypertension  
    M Li, S Vattulainen, J Aho, M Orcholski, V Rojas, K Yuan, VA de Jesus Perez, M Helenius, P Taimen, S Myllykangas, J Koskenvuo, TP Alastalo, Am J Resp Mol Cell Biol, 1/1/2014
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Abstracts/Posters

  • Whole exome sequencing leads to discovery of novel gene variants in PAH
    VA de Jesus Perez, AHA Scientific Sessions, 1/1/2012
  • Recruitment of Disheveled by Integrin Linked Kinase 1 Regulates Bone Morphogenetic Protein 2 Induced Pulmonary Smooth Muscle Cell Motility
    VA de Jesus Perez, American Heart Association Scientific Sessions, 1/1/2009
  • Bone Morphogenetic Protein 2 promotes pulmonary artery smooth muscle cell motility via cross-talk with both Wnt/b-catenin and Wnt/RhoA-Rac1 pathways
    Vinicio A. de Jesus Perez and Marlene Rabinovitch, American Thoracic Society Annual Meeting, 1/1/2007
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Lectures

  • Wall Center Video Series “What you need to know about the Pathology of PAH” 
    1/1/2015
  • Whole exome sequencing for the discovery of novel genetic modifiers in PAH Genomic Predisposition and Therapeutic Avenues in Pulmonary Hypertension 
    1/1/2015
  • BMPR2 and Wnt signaling BMPR2 and Pulmonary Hypertension at the Crossroads 
    1/1/2015
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Grant Support

  • Stanford Undergraduate URM Summer Cardiovascular Research ProgramSTANFORD UNIVERSITY2019–2029
  • The Role of the CES1 in the Pathogenesis of Pulmonary Arterial HypertensionSTANFORD UNIVERSITY2024–2028
  • The Role of the CES1 in the Pathogenesis of Pulmonary Arterial HypertensionSTANFORD UNIVERSITY2024–2028
  • The Role of the CES1 in the Pathogenesis of Pulmonary Arterial HypertensionSTANFORD UNIVERSITY2024–2028
  • Endothelial-pericyte interactions in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026
  • The Wnt7a/ROR2 axis in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026
  • Endothelial-pericyte interactions in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026
  • Endothelial-pericyte interactions in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026
  • The Wnt7a/ROR2 axis in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026
  • The Wnt7a/ROR2 axis in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026
  • The Wnt7a/ROR2 axis in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026
  • The Wnt7a/ROR2 axis in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026
  • The Wnt7a/ROR2 axis in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026
  • The Wnt7a/ROR2 axis in the pathogenesis of pulmonary arterial hypertensionSTANFORD UNIVERSITY2017–2026

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